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Genetic susceptibility mechanisms. Most susceptibility loci map to non-coding regions on the genome, are primarily situated in Energetic promoters or enhancers, and modify the binding web-sites of a variety of transcription elements.
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Environmental or self-antigens and homotypic interactions set off BCR and Toll-like receptor (TLR) signaling, amplifying the reaction of CLL cells to other indicators in the microenvironment and increasing the activation of anti-apoptotic and proliferation pathways.
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